vitro primary human coronary artery smooth muscle cells hcsmcs (Cell Applications Inc)
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vitro primary human coronary artery smooth muscle cells hcsmcs
Vitro Primary Human Coronary Artery Smooth Muscle Cells Hcsmcs, supplied by Cell Applications Inc, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/vitro+primary+human+coronary+artery+smooth+muscle+cells+hcsmcs/Human+Coronary+Artery+Smooth+Muscle+Cells%3A+Plaque%3A+HCASMC-q/pm36306211-45-29-41
Average 92 stars, based on 1 article reviews
Vitro Primary Human Coronary Artery Smooth Muscle Cells Hcsmcs, supplied by Cell Applications Inc, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/vitro+primary+human+coronary+artery+smooth+muscle+cells+hcsmcs/Human+Coronary+Artery+Smooth+Muscle+Cells%3A+Plaque%3A+HCASMC-q/pm36306211-45-29-41
Average 92 stars, based on 1 article reviews
vitro primary human coronary artery smooth muscle cells hcsmcs - by Bioz Stars,
2026-10
92/100 stars
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Knockdown:Article Title: Smooth muscle cell FTO regulates contractile function. Article Snippet: The fat mass and obesity gene (FTO) is a N-methyladenosine RNA demethylase that was initially linked by Genome-wide association studies to increased rates of obesity.. Subsequent studies have revealed multiple mass-independent effects of the gene, including cardiac myocyte contractility.. We created a mouse with a conditional and inducible smooth muscle cell deletion of Fto (Myh11 Creþ Fto) and did not observe any changes in mouse body mass or mitochondrial metabolism. In Vitro:Article Title: Smooth muscle cell FTO regulates contractile function. Article Snippet: The fat mass and obesity gene (FTO) is a N-methyladenosine RNA demethylase that was initially linked by Genome-wide association studies to increased rates of obesity.. Subsequent studies have revealed multiple mass-independent effects of the gene, including cardiac myocyte contractility.. We created a mouse with a conditional and inducible smooth muscle cell deletion of Fto (Myh11 Creþ Fto) and did not observe any changes in mouse body mass or mitochondrial metabolism. |